CORNEAL GRAFT REJECTION: MECHANISM, CLINICAL MANIFESTATIONS, DIAGNOSIS, AND TREATMENT

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JAVADI M.A.

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Journal: BINA Year:2004 | Volume:10 | Issue:1 Page(s): 90-105

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Information Journal Paper

Title

CORNEAL GRAFT REJECTION: MECHANISM, CLINICAL MANIFESTATIONS, DIAGNOSIS, AND TREATMENT

Author(s)

JAVADI M.A. | Issue Writer Certificate

Keywords

CORNEAL TRANSPLANTATIONQ3

REJECTIONQ3

DIAGNOSISQ3

TREATMENTQ3

Abstract

CORNEAL TRANSPLANTATION is the most common and successful form of solid organ transplantation with a 2-year survival rate of over 90% in low risk cases. This high success rate may be due to the immune privileged status of the cornea, ACAID (anterior chamber associated immune deviation) induction phenomena and secretion of inhibitory molecules by corneal cells such as IL1-ra, IL-I a and Fas ligand. However, immunological REJECTION is the leading cause of corneal graft failure. This process is primarily mediated by CD4+ T cells of the Th1 phenotype. The mainstay of corneal graft REJECTION TREATMENT is topical and systemic steroid. Recently topical and systemic cyclosporine A has also yielded promising results. This article reviews the molecular mechanisms of immunity in corneal graft REJECTION in addition to its prevention and TREATMENT. Correct preoperative case selection is vital in preventing graft REJECTION.

Cites

TOPICAL CYCLOSPORINE A 2% IN MANAGEMENT OF ENDOTHELIAL CORNEAL ALLOGRAFT REJECTION

References

بانک چشم

Cite

APA: Copy

JAVADI, M.A.. (2004). CORNEAL GRAFT REJECTION: MECHANISM, CLINICAL MANIFESTATIONS, DIAGNOSIS, AND TREATMENT. BINA, 10(1), 90-105. SID. https://sid.ir/paper/42813/en

Vancouver: Copy

JAVADI M.A.. CORNEAL GRAFT REJECTION: MECHANISM, CLINICAL MANIFESTATIONS, DIAGNOSIS, AND TREATMENT. BINA[Internet]. 2004;10(1):90-105. Available from: https://sid.ir/paper/42813/en

IEEE: Copy

M.A. JAVADI, “CORNEAL GRAFT REJECTION: MECHANISM, CLINICAL MANIFESTATIONS, DIAGNOSIS, AND TREATMENT,” BINA, vol. 10, no. 1, pp. 90–105, 2004, [Online]. Available: https://sid.ir/paper/42813/en

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