Information Journal Paper
APA:
CopyKANN, S., STES, C., REICHARD, J.F., HUANG, M.Y., SARTOR, M.A., & SCHWEMBERGER, S.. (2005). BUTILHYDROQUINONE PROTECTS CELLS GENETICALLY DEFICIENT IN GLUTATHIONE BIOSYNTHESIS FROM ARSENITE-INDUCED APOPTOSIS WITHOUT SIGNIFICANTLY CHANGING THEIR PROOXIDANT STATUS. TOXICOLOGICAL SCIENCES, 87(2), 365-384. SID. https://sid.ir/paper/588232/en
Vancouver:
CopyKANN S., STES C., REICHARD J.F., HUANG M.Y., SARTOR M.A., SCHWEMBERGER S.. BUTILHYDROQUINONE PROTECTS CELLS GENETICALLY DEFICIENT IN GLUTATHIONE BIOSYNTHESIS FROM ARSENITE-INDUCED APOPTOSIS WITHOUT SIGNIFICANTLY CHANGING THEIR PROOXIDANT STATUS. TOXICOLOGICAL SCIENCES[Internet]. 2005;87(2):365-384. Available from: https://sid.ir/paper/588232/en
IEEE:
CopyS. KANN, C. STES, J.F. REICHARD, M.Y. HUANG, M.A. SARTOR, and S. SCHWEMBERGER, “BUTILHYDROQUINONE PROTECTS CELLS GENETICALLY DEFICIENT IN GLUTATHIONE BIOSYNTHESIS FROM ARSENITE-INDUCED APOPTOSIS WITHOUT SIGNIFICANTLY CHANGING THEIR PROOXIDANT STATUS,” TOXICOLOGICAL SCIENCES, vol. 87, no. 2, pp. 365–384, 2005, [Online]. Available: https://sid.ir/paper/588232/en