INTERACTION BETWEEN AT1 RECEPTOR AND NF-ΚB IN HYPOTHALAMIC PARAVENTRICULAR NUCLEUS CONTRIBUTES TO OXIDATIVE STRESS AND SYMPATHOEXCITATION BY MODULATING NEUROTRANSMITTERS IN HEART FAILURE

YU, X.J., & SUO, Y.P.. (2013). INTERACTION BETWEEN AT1 RECEPTOR AND NF-?B IN HYPOTHALAMIC PARAVENTRICULAR NUCLEUS CONTRIBUTES TO OXIDATIVE STRESS AND SYMPATHOEXCITATION BY MODULATING NEUROTRANSMITTERS IN HEART FAILURE. CARDIOVASCULAR TOXICOLOGY, 13(4), 381-390. SID. https://sid.ir/paper/671418/en

YU X.J., SUO Y.P.. INTERACTION BETWEEN AT1 RECEPTOR AND NF-?B IN HYPOTHALAMIC PARAVENTRICULAR NUCLEUS CONTRIBUTES TO OXIDATIVE STRESS AND SYMPATHOEXCITATION BY MODULATING NEUROTRANSMITTERS IN HEART FAILURE. CARDIOVASCULAR TOXICOLOGY[Internet]. 2013;13(4):381-390. Available from: https://sid.ir/paper/671418/en

X.J. YU, and Y.P. SUO, “INTERACTION BETWEEN AT1 RECEPTOR AND NF-?B IN HYPOTHALAMIC PARAVENTRICULAR NUCLEUS CONTRIBUTES TO OXIDATIVE STRESS AND SYMPATHOEXCITATION BY MODULATING NEUROTRANSMITTERS IN HEART FAILURE,” CARDIOVASCULAR TOXICOLOGY, vol. 13, no. 4, pp. 381–390, 2013, [Online]. Available: https://sid.ir/paper/671418/en