In this paper, we have punctured unit Zq-simplex code and constructed a new code called unit Zq-simplex code of type _. In particular, we find the parameters of these codes and have proved that it is an [Φ, (q) + 2, 2, Φ, (q) + 2-Φ, (q) / Φ, (p)] Zq-linear code if k = 2 and [Φ, (q)k-1/ Φ, (q)-1 + Φ, (q)k-2, k, Φ, (q)k-1/ Φ, (q)-1 + Φ, (q)k-2 –,(Φ, (q) / Φ, (p)) (Φ, (q)k-1 –,1/ Φ, (q)-1 + Φ, (q)k-3)] Zq-linear code if k ≥,3,where p is the smallest prime divisor of q: For q is a prime power and rank k = 3,we have given the weight distribution of unit Zq-simplex Codes of type α, . Also, we have introduced some new code from Zq-simplex code called zero divisor Zq-simplex code and proved that it is an [ρ, k-1/ ρ,-1, k, ρ, k-1/ ρ,-1 –,(ρ, (k-1) –,1/ ρ,-1) (q/p)] Zq-linear code, where ρ,= q-Φ,(q) and p is the smallest prime divisor of q: Further, we obtain weight distribution of zero divisor Zq-simplex code for rank k = 3 and q is a prime power.

Meat is considered an excellent and nutritious source of protein. However, due to the high cost of this animal protein and economic profit access of some producers, frauds like usage meat species of commercially lower value are noticeable. The aim of this study is to identify low-cost protein frauds such as chicken meat by the molecular method. DNA was extracted from 10 kinds of industrial premium beef burgers, and control samples involved raw beef and chicken meat. Then, simplex-PCR and duplex-PCR were run on the extracted DNA using specific primers of cytochrome b gene in beef and 12s rRNA gene in chicken. Specific genes of beef cytochrome b and chicken 12s rRNA amplified 274 bp and 183 bp fragments respectively in all premium burger samples in simplex-PCR and duplex-PCR. Amplification of fragments of 183 bp by simplex and duplex-PCR suggests adulteration of premium hamburgers by partial replacement of beef with chicken.

Introduction: The goal of this study is to determine the prevalence of ocular herpes simplex manifestations between September 2004-March 2005. The importance of this study is in reducing symptoms and complications of herpes simplex keratitis (Such as iritis, blindness, and decreased visual acuity due to corneal scarring, perforated corneal ulcer, secondary infection with bacteria or fungi, secondary glaucoma) and reducing the risk of recurrence and improving corneal graft survival after penetrating keratoplasty.Materials & Methods: Sampling procedure was sequential. Forty nine patients were selected from the ophthalmic clinics who had ocular herpes simplex manifestation and were examined by completing questionnaires.Results: The results of this study showed that the prevalence rate of epithelial keratitits was 65.3%, stromal keratitis %48, conjunctivitis 4.1%, uveitis 4.1%. The most trigger factors were as follow: Psychological stress 34.7%; sunlight 8.2%; fever 4.1%, local trauma 4.1%; systemic disease 2.05%. The prevalence rate of primary infection was 22.4% and the recurrent infection was 77.6%. Forty seven percent of these patients had visual acuity less than 4.10.Conclusion: Because the rate of corneal scorring increases with any episodic disease recurrence, the necessity of prevention of disease recurrence increases. It is necessary to educate people to recognize the disease manifestations, provocation factors, and preventing methods.

Herpes simplex encephalitis (HSE) is a life threatening outcome of Herpes simplex virus (HSV) infection of the central nervous system (CNS). HSV accounts for 5-2 percent of all cases of encephalitis.One third of cases occur in those younger than 20 years old and one half in those older than 50 years old.Clinical diagnosis is recommended in the encephalopathic, febrile patients with focal neurological signs. However, the clinical findings are not pathogonomic because numerous other diseases of CNS can mimic HSE.Diagnosis should be confirmed based on medical history, analysis of cerebrospinal fluid (CSF) for protein and glucose contents, the cellular analysis and identifying the pathogens by serology and Polymerase Chain Reaction (PCR) amplification.The diagnostic gold standard is the detection of HSV DNA in the cerebrospinal fluid by PCR. But negative results need to be interpreted regarding the patients clinical signs and symptoms and the time of CSF sampling. Spike and slow wave patterns is observed in Electroencephalogram (EEG). Neuroimaging, especially Magnetic Resonance Imaging (MRI) is essential for evaluating the patients, which shows temporal lobe edema or hemorrhage.All patients with HSE should be treated by intravenous Acyclovir (10 mg/kg q8hr for 14-21 days). After completing therapy, PCR of the CSF can confirm the elimination of replicating virus, assisting further management of the patient.