MODELING ATHLETE’S HEART SYNDROME CAUSED BY HYPERTENSION IN STRENGTH TRAINING

Q: How can I download an article?

To download an article from SID, first log in to the site, search for the article title, and click on the 'Download Article' option.

Q: How can I download an ISI article?

To download an ISI article on SID, enter the keyword or article title in the search bar, view the relevant results, click on the desired article, and select the 'Download Article' option.

Q: How can I access the SID database?

To access the SID database, visit SID.ir, create an account, and log in to access scientific resources.

Q: Is downloading articles from SID free?

Some articles on SID are available for free, while others require payment. Details are specified on the article's page.

TORBATI SAEED; DANESHMEHR ALIREZA

مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

Journal Paper

Paper Information

Journal: Modares Mechanical Engineering Year:2018 | Volume:18 | Issue:3 Page(s): 389-394

Download Full-Text

Persian Verion

View:

1,408

Download:

Cites:

Information Journal Paper

Title

MODELING ATHLETE’S HEART SYNDROME CAUSED BY HYPERTENSION IN STRENGTH TRAINING

Author(s)

TORBATI SAEED | DANESHMEHR ALIREZA | Issue Writer Certificate

Keywords

ATHLETE’S HEART SYNDROMEQ1

FINITE GROWTHQ1

CONTINUUM MECHANICSQ1

STEM CELLSQ1

Abstract

Persistent strength training can increase ventricular blood pressure and volume and the resultant loading in ventricles of the human heart. It is proved that pressure overload can increase ventricular thickness and volume. In this article, we modeled ATHLETE’S HEART SYNDROME macroscopically arising from pressure overload using CONTINUUM MECHANICS and finite elements methods. We tried to improve previous results by using a more precise geometry and loading and by modifying previous equations. Firstly, we saw that because the left ventricular pressure was more than the right ventricular pressure, increase in myocardium thickness started from the left ventricle and secondly, this increase in myocardium thickness started from lower regions that located far from the right ventricle. Then, it was shown that thicker regions with greater values of the growth multiplier had less stress than regions with less values of the growth multiplier. As time passed and more loading cycles were applied to the endocardium, myocardium thickness increased gradually until the growth multiplier reached its maximum threshold value. Finally, we demonstrated that when the ventricular pressure rises and hypertrophy occurs, residual stresses remain in the myocardium after unloading.

Cites

No record.

References

No record.

Cite

APA: Copy

TORBATI, SAEED, & DANESHMEHR, ALIREZA. (2018). MODELING ATHLETE’S HEART SYNDROME CAUSED BY HYPERTENSION IN STRENGTH TRAINING. MODARES MECHANICAL ENGINEERING, 18(3 ), 389-394. SID. https://sid.ir/paper/178971/en

Vancouver: Copy

TORBATI SAEED, DANESHMEHR ALIREZA. MODELING ATHLETE’S HEART SYNDROME CAUSED BY HYPERTENSION IN STRENGTH TRAINING. MODARES MECHANICAL ENGINEERING[Internet]. 2018;18(3 ):389-394. Available from: https://sid.ir/paper/178971/en

IEEE: Copy

SAEED TORBATI, and ALIREZA DANESHMEHR, “MODELING ATHLETE’S HEART SYNDROME CAUSED BY HYPERTENSION IN STRENGTH TRAINING,” MODARES MECHANICAL ENGINEERING, vol. 18, no. 3 , pp. 389–394, 2018, [Online]. Available: https://sid.ir/paper/178971/en

Related Journal Papers