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Information Journal Paper

Title

MITOPTOSIS, A NOVEL MITOCHONDRIAL DEATH MECHANISM LEADING PREDOMINANTLY TO ACTIVATION OF AUTOPHAGY

Pages

  1-3

Abstract

 Sometimes some members of multicellular organisms need to sacrifice for the good of the whole. Perhaps with the exception of immunomodulatory processes (1, 2), it is the intrinsic death pathway, often triggered by p53 (3-5), modulated by Bcl2-family members, and executed primarily by caspases that is most commonly employed to trigger cell death (6-8). Apoptotic or autophagic cell death is triggered by physical insults such as cold (9), natural compounds and their derivatives (10-12), viruses (13), or even disturbances within the cell cycle (14, 15). Apoptotic cell death is preceded by mitochondrial release of cytochrome c, which leads to increases in cytochrome c in serum (16).

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    APA: Copy

    JANGAMREDDY, JAGANMOHAN REDDY, & LOS, MAREK J.. (2012). MITOPTOSIS, A NOVEL MITOCHONDRIAL DEATH MECHANISM LEADING PREDOMINANTLY TO ACTIVATION OF AUTOPHAGY. HEPATITIS MONTHLY, 12(8), 1-3. SID. https://sid.ir/paper/306799/en

    Vancouver: Copy

    JANGAMREDDY JAGANMOHAN REDDY, LOS MAREK J.. MITOPTOSIS, A NOVEL MITOCHONDRIAL DEATH MECHANISM LEADING PREDOMINANTLY TO ACTIVATION OF AUTOPHAGY. HEPATITIS MONTHLY[Internet]. 2012;12(8):1-3. Available from: https://sid.ir/paper/306799/en

    IEEE: Copy

    JAGANMOHAN REDDY JANGAMREDDY, and MAREK J. LOS, “MITOPTOSIS, A NOVEL MITOCHONDRIAL DEATH MECHANISM LEADING PREDOMINANTLY TO ACTIVATION OF AUTOPHAGY,” HEPATITIS MONTHLY, vol. 12, no. 8, pp. 1–3, 2012, [Online]. Available: https://sid.ir/paper/306799/en

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