مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

video

مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

sound

مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

Persian Version

مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

View:

360
مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

Download:

0
مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

Cites:

Information Journal Paper

Title

APOPTOSIS: PROGRAMMED CELL DEATH

Pages

  48-57

Abstract

APOPTOSIS or PROGRAMMED CELL DEATH is known as a conserved gene-directed mechanism for the specific elimination of unnecessary or unwanted cells from an organism and is involved in many immune system mechanisms and diseases. The main differences of this pathway with cell necrosis as two main pathways for elimination of unwanted cells are the absence of inflammation and the restricted effect on target cells. APOPTOSIS has an important role in biological processes such as normal development, tissue homeostasis, elimination of destroyed cells or virus infected cells and remove of self antigen-activated immune cells. APOPTOSIS is important for regulation of cell growth and proliferation, development and body health and many autoimmune diseases, cancers and viral infections are the result of impaired or inhibited PROGRAMMED CELL DEATH. Therefore the main objective of APOPTOSIS research is to identify molecular components and regulatory mechanisms specially Bcl-2 and IAP FAMILY as the most important regulators of APOPTOSIS and this information may lead to application of therapeutic agents that can modulate this process in the treatment of neurodegenerative disorders and proliferative diseases such as cancer.

Cites

  • No record.
  • References

  • No record.
  • Cite

    APA: Copy

    HONARDOOST, M., SOLEIMANJAHI, H., & RAJAEI, F.. (2013). APOPTOSIS: PROGRAMMED CELL DEATH. JOURNAL OF INFLAMMATORY DISEASES (THE JOURNAL OF QAZVIN UNIVERSITY OF MEDICAL SCIENCES), 17(3 (68)), 48-57. SID. https://sid.ir/paper/624611/en

    Vancouver: Copy

    HONARDOOST M., SOLEIMANJAHI H., RAJAEI F.. APOPTOSIS: PROGRAMMED CELL DEATH. JOURNAL OF INFLAMMATORY DISEASES (THE JOURNAL OF QAZVIN UNIVERSITY OF MEDICAL SCIENCES)[Internet]. 2013;17(3 (68)):48-57. Available from: https://sid.ir/paper/624611/en

    IEEE: Copy

    M. HONARDOOST, H. SOLEIMANJAHI, and F. RAJAEI, “APOPTOSIS: PROGRAMMED CELL DEATH,” JOURNAL OF INFLAMMATORY DISEASES (THE JOURNAL OF QAZVIN UNIVERSITY OF MEDICAL SCIENCES), vol. 17, no. 3 (68), pp. 48–57, 2013, [Online]. Available: https://sid.ir/paper/624611/en

    Related Journal Papers

  • No record.
  • Related Seminar Papers

  • No record.
  • Related Plans

  • No record.
  • Recommended Workshops






    مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources
    مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources
    مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources
    مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources
    مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources
    مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources
    مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources
    File Not Exists.
    Move to top
    telegram sharing button
    whatsapp sharing button
    linkedin sharing button
    twitter sharing button
    email sharing button
    email sharing button
    email sharing button
    sharethis sharing button