مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Information Journal Paper

Title

Genetic and Epigenetic of Medullary Thyroid Cancer

Pages

  142-150

Abstract

 Medullary Thyroid carcinoma (MTC) is an infrequent calcitonin-producing neuroendocrine tumor that initiates from the parafollicular C cells of the thyroid gland. Several genetic and epigenetic alterations are collaterally responsible for medullary thyroid carcinogenesis. In this review article, we shed light on all the genetic and epigenetic hallmarks of MTC. From the genetic perspective, RET, HRAS, and KRAS are the most important genes that are characterized in MTC. From the epigenetic perspective, Ras-association domain family member 1A, telomerase reverse transcriptase promoter methylations, overexpression of histone methyltransferases, EZH2 and SMYD3, and wide ranging increase and decrease in non-coding RNAs can be responsible for medullary thyroid carcinogenesis.

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    • Cite

    APA: Copy

    KHATAMI, FATEMEH, & TAVANGAR, SEYED MOHAMMAD. (2018). Genetic and Epigenetic of Medullary Thyroid Cancer. IRANIAN BIOMEDICAL JOURNAL, 22(3), 142-150. SID. https://sid.ir/paper/749832/en

    Vancouver: Copy

    KHATAMI FATEMEH, TAVANGAR SEYED MOHAMMAD. Genetic and Epigenetic of Medullary Thyroid Cancer. IRANIAN BIOMEDICAL JOURNAL[Internet]. 2018;22(3):142-150. Available from: https://sid.ir/paper/749832/en

    IEEE: Copy

    FATEMEH KHATAMI, and SEYED MOHAMMAD TAVANGAR, “Genetic and Epigenetic of Medullary Thyroid Cancer,” IRANIAN BIOMEDICAL JOURNAL, vol. 22, no. 3, pp. 142–150, 2018, [Online]. Available: https://sid.ir/paper/749832/en

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