مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Information Journal Paper

Title

HOST-MICROBIOTA INTERACTION IS MYD88-INDEPENDENT IN THE INTESTINAL TRACT UNDER PHYSIOLOGIC CONDITION (LETTER TO THE EDITOR)

Pages

  210-211

Abstract

 The role of microbiota in health and disease is the subject of rigorous investigation. Several studies have demonstrated that microbiota and the pattern-recognition receptors contribute to intestinal tumourigenesis, the exact mechanism of which is still obscure. MyD88 is the downstream effector of all TOLL-LIKE RECEPTORS (TLRs) except TLR3. However, the alternative MyD88-independent pathway is functional downstream of not only TLR3, but also TLR1.2, 2.6, 4, and 5. TLR4 stimulation with intraperitoneal lipopolysaccharide exerts distinct functional effect on the intestinal motility via MyD88-dependent and-independent pathways.

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    • Cite

    APA: Copy

    MOOSSAVI, SHIRIN, & REZAEI, NIMA. (2012). HOST-MICROBIOTA INTERACTION IS MYD88-INDEPENDENT IN THE INTESTINAL TRACT UNDER PHYSIOLOGIC CONDITION (LETTER TO THE EDITOR). AVICENNA JOURNAL OF MEDICAL BIOTECHNOLOGY (AJMB), 4(4 (15)), 210-211. SID. https://sid.ir/paper/313794/en

    Vancouver: Copy

    MOOSSAVI SHIRIN, REZAEI NIMA. HOST-MICROBIOTA INTERACTION IS MYD88-INDEPENDENT IN THE INTESTINAL TRACT UNDER PHYSIOLOGIC CONDITION (LETTER TO THE EDITOR). AVICENNA JOURNAL OF MEDICAL BIOTECHNOLOGY (AJMB)[Internet]. 2012;4(4 (15)):210-211. Available from: https://sid.ir/paper/313794/en

    IEEE: Copy

    SHIRIN MOOSSAVI, and NIMA REZAEI, “HOST-MICROBIOTA INTERACTION IS MYD88-INDEPENDENT IN THE INTESTINAL TRACT UNDER PHYSIOLOGIC CONDITION (LETTER TO THE EDITOR),” AVICENNA JOURNAL OF MEDICAL BIOTECHNOLOGY (AJMB), vol. 4, no. 4 (15), pp. 210–211, 2012, [Online]. Available: https://sid.ir/paper/313794/en

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