CASE REPORT: CD 25/26(+T), RARE MUTATION IN BETA-GLOBIN GENE

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HOSSEINPOUR FEYZI A.A.; HOSSEINPOUR FEYZI M.A.; POULADI N.; HAGHI MAHDI; AZARFAM P.

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Journal: MEDICAL JOURNAL OF TABRIZ UNIVERSITY OF MEDICAL SCIENCES Year:2007 | Volume:29 | Issue:3 Page(s): 139-142

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Title

CASE REPORT: CD 25/26(+T), RARE MUTATION IN BETA-GLOBIN GENE

Author(s)

Keywords

CD 25/26 (+T)Q3

BETA-THALASSEMIAQ3

HAEMOGLOBINQ3

Abstract

b-thalassemia is a heterogenous group of genetic alteration characterized by a deficient synthesis (b+) or an absent (bo) of b- globin chains. However, between these two clinical forms there are a wide range of clinical phenotypes. We reported a very rare bo thalassmia mutation, Cd25/26(+T), in a family with normal body structure and transfusion dependent from Azerbaijan-Iran for the first time. The comparison of hemoglobin level revealed that they have a high level HbF (98%) that help their normal growth.

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APA: Copy

HOSSEINPOUR FEYZI, A.A., HOSSEINPOUR FEYZI, M.A., POULADI, N., HAGHI, MAHDI, & AZARFAM, P.. (2007). CASE REPORT: CD 25/26(+T), RARE MUTATION IN BETA-GLOBIN GENE. MEDICAL JOURNAL OF TABRIZ UNIVERSITY OF MEDICAL SCIENCES, 29(3), 139-142. SID. https://sid.ir/paper/47180/en

Vancouver: Copy

HOSSEINPOUR FEYZI A.A., HOSSEINPOUR FEYZI M.A., POULADI N., HAGHI MAHDI, AZARFAM P.. CASE REPORT: CD 25/26(+T), RARE MUTATION IN BETA-GLOBIN GENE. MEDICAL JOURNAL OF TABRIZ UNIVERSITY OF MEDICAL SCIENCES[Internet]. 2007;29(3):139-142. Available from: https://sid.ir/paper/47180/en

IEEE: Copy

A.A. HOSSEINPOUR FEYZI, M.A. HOSSEINPOUR FEYZI, N. POULADI, MAHDI HAGHI, and P. AZARFAM, “CASE REPORT: CD 25/26(+T), RARE MUTATION IN BETA-GLOBIN GENE,” MEDICAL JOURNAL OF TABRIZ UNIVERSITY OF MEDICAL SCIENCES, vol. 29, no. 3, pp. 139–142, 2007, [Online]. Available: https://sid.ir/paper/47180/en

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