مرکز اطلاعات علمی Scientific Information Database (SID) - Trusted Source for Research and Academic Resources

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Cites:

1

Information Journal Paper

Title

The role of human T-lymphotropic virus (HTLV) in cardiovascular diseases: A review of literature

Pages

  183-187

Abstract

Cardiovascular Diseases are a major cause of morbidity and mortality. Chronic inflammation is an important risk factor for Atherosclerosis, and viral infections can cause Cardiovascular Disease by developing inflammation. Infection with human T-lymphotropic virus (HTLV) is endemic in some parts of the world such as Japan, Africa, Caribbean islands, South America, and Iran. HTLV-1 is an oncogenic retrovirus, and can cause adult T-cell leukemia/lymphoma (ATL or ATLL). It also causes HTLV-I-associated myelopathy/tropical spastic paraparesis (HAM/TSP). A number of inflammatory diseases such as uveitis, arthritis, and Sjogren's syndrome are also associated with the virus. A few case reports have shown the direct involvement of the heart in HTLV-1-positive patients who develop ATLL. The purpose of this study was to review the literature relevant with the role of HTLV in Cardiovascular Diseases.

Cites

References

Cite

APA: Copy

Abolbashari, Samaneh, GHAYOUR MOBARHAN, MAJID, EBRAHIMI, MAHMOUD, & MESHKAT, ZAHRA. (2018). The role of human T-lymphotropic virus (HTLV) in cardiovascular diseases: A review of literature. ARYA ATHEROSCLEROSIS, 14(4), 183-187. SID. https://sid.ir/paper/732030/en

Vancouver: Copy

Abolbashari Samaneh, GHAYOUR MOBARHAN MAJID, EBRAHIMI MAHMOUD, MESHKAT ZAHRA. The role of human T-lymphotropic virus (HTLV) in cardiovascular diseases: A review of literature. ARYA ATHEROSCLEROSIS[Internet]. 2018;14(4):183-187. Available from: https://sid.ir/paper/732030/en

IEEE: Copy

Samaneh Abolbashari, MAJID GHAYOUR MOBARHAN, MAHMOUD EBRAHIMI, and ZAHRA MESHKAT, “The role of human T-lymphotropic virus (HTLV) in cardiovascular diseases: A review of literature,” ARYA ATHEROSCLEROSIS, vol. 14, no. 4, pp. 183–187, 2018, [Online]. Available: https://sid.ir/paper/732030/en

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